Wi-fi gadgets linked to cancer risk – study

NZ Herald | 2 Aug 2015

Mobile phones, laptops and wi-fi may increase your chance of cancer and other brain diseases, researchers warn.

Supporting claims that wireless devices carry a health risk, a study showed they may help cause a metabolic imbalance linked to brain tumours and other neurological disorders including Parkinson’s and Alzheimer’s.

But other experts insisted that while the science in the study was sound, the risk to human health was still tiny and cautioned against an alarmist reaction to the findings.

The research, published in the Electromagnetic Biology & Medicine Journal, was led by Dr Igor Yakymenko from Kiev.

It looked at the effect of low-intensity radiofrequency radiation (RFR) emitted by wireless devices, using studies from the US, Finland and Ukraine that mostly examined the effect of RFR on rats.

Dr Yakymenko said there was evidence that four years of using a mobile phone for at least an hour a day increased the incidence of some rare brain tumours by three to five times. The amount of RFR we are routinely exposed to has increased 5,000 times in the past 20 years and although health risks are low, ailments can take up to 30 years to develop, he added.

The study suggested keeping mobile phone use to a minimum and always using headphones. But Sarah Williams, health information manager at Cancer Research UK, said: “It’s unlikely that using mobile phones causes brain tumours. Over the last two decades, mobile phone use has rocketed in the UK but we haven’t seen any similar rise in the rates of brain tumours.” (Study Abstract below)

– Daily Mail

Oxidative mechanisms of biological activity of low-intensity radiofrequency radiation

Abstract

This review aims to cover experimental data on oxidative effects of low-intensity radiofrequency radiation (RFR) in living cells. Analysis of the currently available peer-reviewed scientific literature reveals molecular effects induced by low-intensity RFR in living cells; this includes significant activation of key pathways generating reactive oxygen species (ROS), activation of peroxidation, oxidative damage of DNA and changes in the activity of antioxidant enzymes. It indicates that among 100 currently available peer-reviewed studies dealing with oxidative effects of low-intensity RFR, in general, 93 confirmed that RFR induces oxidative effects in biological systems. A wide pathogenic potential of the induced ROS and their involvement in cell signaling pathways explains a range of biological/health effects of low-intensity RFR, which include both cancer and non-cancer pathologies. In conclusion, our analysis demonstrates that low-intensity RFR is an expressive oxidative agent for living cells with a high pathogenic potential and that the oxidative stress induced by RFR exposure should be recognized as one of the primary mechanisms of the biological activity of this kind of radiation.

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1Institute of Experimental Pathology, Oncology and Radiobiology of NAS of Ukraine,Kyiv, Ukraine,
2Department of Biophysics, Bila Tserkva National Agrarian University,Bila Tserkva,Ukraine,
3School of Public and Environmental Affairs, Indiana University Bloomington,Bloomington, IN, USA,
4A.I.Virtanen Institute, University of Eastern Finland,Kuopio, Finland, and
5Department of Structural and Functional Biology, University of Campinas,Campinas, SP, Brazil
Address correspondence to Prof. Igor Yakymenko, Laboratory of Biophysics, Institute of Experimental Pathology, Oncology and Radiobiology of NAS of Ukraine, Vasylkivska str. 45, Kyiv, 03022Ukraine. E-mail:

Read More: http://informahealthcare.com/doi/abs/10.3109/15368378.2015.1043557

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